Schematic model depicting the role of SlNAC2 in regulating saline–alkali stress tolerance in tomato. (IMAGE)

Nanjing Agricultural University The Academy of Science

Schematic model depicting the role of SlNAC2 in regulating saline–alkali stress tolerance in tomato.

Caption

Schematic model depicting the role of SlNAC2 in regulating saline–alkali stress tolerance in tomato. Following exposure to saline–alkali stress, SlNAC2 is upregulated and directly targets the SlCV promoter, leading to transcriptional activation of SlCV and increased ROS levels. In addition, SlNAC2 interacts with the promoters of SlCOMT2 and SlSNAT, resulting in transcriptional repression and a subsequent decline in endogenous MT content. The reduction in MT further enhances tomato sensitivity to salt–alkali stress. Furthermore, SlDREB2 physically interacts with SlNAC2 and modulates its transcriptional activity, reinforcing that the SlDREB2- SlNAC2 module acts as a central hub controlling saline–alkali stress responses by precisely regulating downstream genes involved in MT biosynthesis and ROS homeostasis.

Credit

Horticulture Research

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