News Release

Mesenchymal stromal cell therapy as breakthrough for rheumatoid arthritis

A new review establishes MSC therapy as a transformative rheumatoid arthritis solution with validated long-term efficacy and safety via preclinical and clinical evidence

Peer-Reviewed Publication

Chinese Medical Journals Publishing House Co., Ltd.

Characteristics of MSCs and their and therapeutic mechanisms in rheumatoid arthritis.

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Characteristics of MSCs and their and therapeutic mechanisms in rheumatoid arthritis. MSC, mesenchymal stromal cell; Tfh, T-follicular helper; Th, T helper; Treg, regulatory T cells.

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Credit: Rheumatology & Autoimmunity

Rheumatoid arthritis (RA), a chronic autoimmune disease affecting 0.5% to 1% of the global population, causes erosive joint damage, pain, and systemic complications, often leading to disability. Conventional treatments (nonsteroidal anti-inflammatory drugs, NSAIDs; disease-modifying antirheumatic drugs, DMARDs; biologics) ease symptoms temporarily but carry severe side effects (gastrointestinal harm, infections) and fail to address root causes.  

 

In a new review in the journal Rheumatology & Autoimmunity, researchers led by Dr. Yingjia Chen (Lotus Lake Capital) position mesenchymal stromal cell (MSC) therapy as a transformative RA solution, synthesizing preclinical and clinical evidence of its long-term efficacy and safety.  

 

Why Conventional RA Treatments Fall Short  

RA stems from dysregulated immune responses attacking joints. Traditional options have critical flaws:  

- NSAIDs/glucocorticoids: No joint protection; long-term use risks osteoporosis, bleeding.  

- DMARDs: Liver/kidney toxicity.  

- Biologics: Costly, raise infection/malignancy risks, and lose efficacy over time.  

 

MSCs: Unique Therapeutic Advantages  

Found in umbilical cord, bone marrow, and adipose tissue, MSCs target RA via four key traits:  

1. Immunomodulation: Suppress overactive immune cells, cut pro-inflammatory cytokines (TNF-α, IL-6), and restore balance.  

2. Tissue repair: Differentiate into cartilage/bone cells, secrete growth factors (IGF-1, VEGF) to heal damage.  

3. Low immunogenicity: Safe donor-derived use with minimal rejection risk.  

4. Targeted migration: Home in on inflamed joints via chemokine receptors.  

 

Multiple clinical studies (1-3-year follow-ups included) confirm sustained benefits of MSC therapy for RA patients. For instance, a 3-year study of 64 refractory RA patients by Yongjun Liu team showed MSCs + low-dose DMARDs sustainedly reduced inflammatory markers (ESR, CRP) and improved DAS28/HAQ scores, with no serious adverse events. Additionally, MSC therapy has low infection/allergy rates, no elevated long-term tumor risk, and better safety than traditional immunosuppression.

 

Path to Broad Use  

The review outlines strategies to optimize MSCs, such as:  

- Personalized selection: UC-MSCs for accessibility, bone marrow MSCs for severe damage.   

- Combinations: MSCs + low-dose DMARDs enhance outcomes.  


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