Obesity may influence how breast cancer spreads
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Updates every hour. Last Updated: 22-Jun-2026 06:15 ET (22-Jun-2026 10:15 GMT/UTC)
W. M. Keck Foundation Bridge Funding Initiative grants $600,000 to Salk scientist, graduate student pairings. The three teams will each tackle their own projects, spanning cancer, neuroscience, and genetics. The grants accelerate high-risk, high-reward science that otherwise would be difficult to pursue in today’s science funding landscape.
Transposable elements (TEs), also called transposons, are DNA sequences capable of moving or replicating from one location to another within a genome. While TEs are the most significant fraction of the human genome (approximately 40-50%), only recently have scientists begun to appreciate their impact impact on human diseases from cancer to neurodegenerative disorders.
Usually, our cells keep TEs quiet when we are young, but in a new study from Boston University Chobanian & Avedisian School of Medicine, researchers have discovered that TEs are actively expressed in human brains as large RNAs that can then be processed into small RNAs (18-32 nucleotides long). This finding provides new molecular insights into how our brains age normally, as well as how neurodegenerative disorders can impact these normal patterns of transposon RNA expression.
OSUCCC – James researchers will present new findings at the 2026 American Society of Clinical Oncology (ASCO) annual meeting, including a program that helps patients get donated oral drugs faster, new insights into colon and breast cancer, and ongoing accessibility gaps across communities in clinical trials.
Using catheters coated with specialized carbon nanotubes, MIT researchers developed a new way to monitor bladder cancer patients for tumor recurrence.
Veterans diagnosed with cancer face a higher risk of suicide attempts — especially in the months following diagnosis — and that risk can persist for years, found a large, national study led by Oregon Health & Science University and the Veterans Health Administration.
Researchers at Columbia University Irving Medical Center have identified a gene that drives the development of neuroendocrine prostate cancer (NEPC), an aggressive form of the disease. The study, to be published May 28 in the Journal of Experimental Medicine (JEM), shows that genetic or pharmacological inhibition of Sirtuin 1 prevents the growth of NEPC tumors in mice, and lays the groundwork for future clinical studies aimed at developing new treatments for NEPC in humans.