Gladstone Institutes launches redesigned website, bringing cutting-edge bomedical research into focus
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Updates every hour. Last Updated: 20-Oct-2026 10:16 ET (20-Oct-2026 14:16 GMT/UTC)
Gladstone Institutes today announced the launch of its newly redesigned website, gladstone.org, offering a modernized, accessible platform that brings its biomedical research closer to the public, scientific community, and key partners.
Discover how the St. Jude Lifetime Cohort Study uncovered community-level factors influencing chronic health condition risks, and how that can improve care.
Scientists at the University of Bath (UK) have produced a new azulene-based fluorescent probe that is five times brighter than the original molecule. Their research has uncovered the tiny molecular changes needed to transform both the colour and brightness of the probes, paving the way for more powerful imaging tools for detecting diseases such as Alzheimer's, Parkinson’s and cancer.
Researchers at the University of South Florida, Tampa General Hospital and Hokkaido University have uncovered an important mechanism behind immune evasion in metastatic colorectal cancer (mCRC). The finding challenges a 30-year-old theory in cancer research and identifies a molecular pathway that offers a new target for treatment.
Experts from the University of Nottingham have discovered how a common genetic mutation disrupts the positioning of a key protein during cell division, revealing a new mechanism behind acute myeloid leukaemia and a potential avenue for future treatment.
Acute myeloid leukaemia (AML) is an aggressive blood cancer that disrupts the production of healthy blood cells. Around one-third of patients carry a mutation in a gene called NPM1, which causes its protein to accumulate in the wrong part of the cell and plays a key role in driving the disease.
Until now, research has largely focused on how the mutated protein moves between the cell's nucleus and the surrounding cytoplasm. But a new study, led by Dr Daniel Booth and Dr Claire Seedhouse at the University of Nottingham's School of Medicine, reveals that what happens during cell division itself is also critical.
A team from IRB Barcelona, in collaboration with the Sant Joan de Déu Research Institute and the Pediatric Cancer Center Barcelona at Sant Joan de Déu Hospital, has described a mechanism by which a cancer-related protein can activate regions of DNA that normally remain silenced.
Using the Drosophila fruit fly, the researchers demonstrated that this activation depends on collaboration with another protein, enabling the study of a mechanism that is difficult to isolate in human cells.
The study, published in G3: Genes|Genomes|Genetics, may help explain previous observations in prostate cancer and B-cell acute lymphoblastic leukemia.