Research uncovers how a poet transformed the story of the first “test-tube baby” into a moving tale that helped highlight the women involved.

Interface modification has been demonstrated as an effective means to enhance the performance of perovskite solar cells. However, the effect depends on the anchoring mode and strength of the interfacial molecules, which determines whether long-term robust interface for carrier viaduct can be achieved under operational light illumination. Herein, we select squaric acid (SA) as the interfacial molecule between the perovskite and SnO₂ layer and propose a self-regulated bilateral anchoring strategy. The unique four-membered ring conjugated structure and dicarboxylic acid groups facilitate stable hydrogen bonds and coordination bonds at both SnO₂/SA and SA/PbI₂ interfaces. The self-transforming property of SA enables the dynamic bilateral anchoring at the buried interface, ultimately releasing residual stress and constructing a stable interfacial molecular bridge. The results show that SA molecular bridge not only can effectively inhibit the generation of diverse charged defects but also serves as an effective electron transport pathway, resulting in improved power conversion efficiency (PCE) from 23.19 to 25.50% and excellent stability at the maximum power point. Additionally, the PCEs of the flexible and large-area (1 cm²) devices were increased to 24.92% and 24.01%, respectively, demonstrating the universal applicability of the bilateral anchoring to PSCs based on different substrates and larger area.

Macrophages rely on lysosomes to balance microbial killing with self-protection, generating reactive oxygen and nitrogen species during phagocytosis. Using platinum nanoelectrodes, researchers at Wuhan University tracked these reactive molecules in real time, revealing that lysosomal pH acts as a chemical dial. Acidic conditions favor hydrogen peroxide, while mild alkalinization promotes nitric oxide, peroxynitrite, and nitrite. This precise pH-dependent control shapes immune responses, oxidative stress, and inflammatory signaling, offering insights for therapies targeting macrophage function.