Human telomerase shows selective cross-species activity, revealing limits of animal models
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Updates every hour. Last Updated: 18-May-2026 16:15 ET (18-May-2026 20:15 GMT/UTC)
In addition to immediate health risks, UV radiation also poses indirect hazards: it corrodes surface coatings on exposed objects (e.g., on aircraft and bridges) and attacks the coated materials. The underlying molecular processes (polymer degradation) are extremely complex. Therefore, a consortium coordinated by Fraunhofer IAF is working within the framework of the BMFTR-funded QPolyDeg project to develop novel quantum algorithms for simulating polymer degradation. Quantum chemical calculations are intended to enable more durable coatings for industrial applications.
Researchers from University Hospital Cologne have developed an autonomous agent-based AI system called ‘SPARK’ that acts as a “digital brain” / publication in Nature Medicine
A team of plant researchers at the University of Cologne decodes a key infection strategy of fungal pathogens / Significant findings for disease control, plant protection and food security as well as for medicine / publication in ‘Science Advances’
The Mount Sinai Health System today announced that Lisa M. Satlin, MD, the Herbert H. Lehman Professor of Pediatrics and Chair of the Jack and Lucy Clark Department of Pediatrics at the Icahn School of Medicine at Mount Sinai, and Pediatrician-in-Chief of Mount Sinai Kravis Children’s Hospital, has been named the 2026 recipient of the American Society of Pediatric Nephrology (ASPN) Founders’ Award. The honor recognizes a lifetime of outstanding contributions to pediatric nephrology through research, education, clinical care, and leadership.
A review finds increasing evidence that obesity and Alzheimer’s disease (AD) are biologically linked. The researchers highlight shared early metabolic disruptions involving mitochondrial dysfunction, chronic inflammation and abnormal signaling from fat tissue, which can affect brain health long before symptoms appear. The research also points to the gut-brain axis as a contributor to neurodegeneration. Together, the findings suggest AD risk may begin earlier than previously thought and support a shift toward whole-body, metabolic-focused prevention.
J. Craig Venter, the genomicist whose work redrew the architecture of modern biology, died on 29 April 2026 in San Diego at the age of 79, following complications from treatment of a recently diagnosed cancer. Brain Health, a new peer-reviewed journal launched today by Genomic Press, publishes in its inaugural issue a scientific tribute by Dr. Julio Licinio that foregrounds a part of Venter’s legacy that other obituaries have understandably treated as background: his earliest major methodological breakthrough emerged at the National Institute of Neurological Disorders and Stroke, where he pioneered the expressed sequence tag as a route to rapidly identifying brain-expressed genes. The tribute traces an arc from that neuroscience starting point through the first complete bacterial genome, the parallel pursuit of the human sequence, large-scale ocean metagenomics, and the construction of the first cell controlled by a chemically synthesized genome.
Pneumonia is responsible for a tremendous burden of disease worldwide. In the U.S., it is a leading cause of death due to infection, especially for those of advanced age. For survivors, pneumonia’s lingering effects such as reduced lung function, scarring and new or worsened respiratory issues like asthma or COPD, may accelerate unhealthy aging. While pneumonia is fundamentally a disease of the lung tissue characterized by inflammation and alveolar damage, medical science has historically relied on symptoms, imaging (X-rays), and microbiological cultures (microbe-directed) to classify the disease, rather than analyzing the specific cellular damage and structural changes in the lungs (histopathology) to create personalized treatment subgroups (subphenotyping). In a new study, researchers from Boston University Chobanian & Avedisian School of Medicine have identified seven different forms of pneumonia. This is the first systematic examination of pulmonary histopathology during pneumonia, resulting in a new framework for understanding pneumonia heterogeneity based on cellular resolution of lung biology.